By Rakesh Ranjan, Amita Ranjan
This publication describes intimately a variety of facets of fluoride toxicity in animals. Animals, like humans, be afflicted by the poisonous results of extra fluoride consumption. They express pathological alterations of their enamel and bone, including a marked relief in urge for food, effective and reproductive potentials, which may end up in serious fiscal losses within the dairy undefined. Laboratory and wild animals additionally be afflicted by this disorder. Animal affliction and monetary losses alike may be minimized via early prognosis of the matter and via adopting compatible preventive and healing measures. The publication info the susceptibility of other animal species, very important assets of toxicity, medical signs, pathophysiology, diagnostic equipment, preventive and healing ways. It deals a worthy source for scientists operating within the fields of toxicology, veterinary technology, animal nutrients, and environmental technological know-how, in addition to for public medical experts, animal welfare activists, public health and wellbeing veterinarians, box veterinarians, doctors and all others drawn to the topic.
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Additional info for Fluoride Toxicity in Animals
1998). Fluoride toxicity induces a wide range of structural and functional changes in the renal system. 2 Chronic Toxicity 43 (Jankaurkar 1974) and decreases Na, K, and ATPase activities (Suketa and Terui 1980). The increase in serum urea nitrogen and creatinine is well documented in fluoride toxicosis in humans and animals (Singh and Swarup 1995; Shivashankara et al. 2000). Histopathological observations in fluoride toxicosis include cloudy swelling, degeneration of tubular epithelia, centrilobular necrosis, atrophy of glomeruli, periglomerular fibrosis, and tubular necrosis (Shashi et al.
WHO regional office for Europe, World Health Organization, Copenhagen Zumpt I (1975) Chronic fluoride poisoning in sheep. J S Afr Vet Assoc 46:161–163 Chapter 3 Fluoride Kinetics and Metabolism Abstract Fluoride, after absorption from the gastrointestinal tract, respiratory tract, or skin and mucous membrane reaches different organs and body tissues through blood circulation. Following oral intake, unabsorbed fluoride is excreted through feces, and 50–70 % of absorbed fluoride is excreted through urine, perspiration, saliva, milk, and egg (in birds) and rest is retained in the body.
High F accumulation in the pineal gland may result in low circulating melatonin and accelerated sexual maturity in females, as observed in an experimental study in gerbils (Luke 1997). 7 mg/kg (Jedra et al. 2001). In the common carp (Cyprinus carpio), fluoride accumulation was highest in the gills, followed by the liver, brain, kidney, muscle, and intestine after exposure to high fluoride concentration in water for 90 days (Cao et al. 2013). 2. 6 to 26 mg/kg wet weight (Lall 1994; Camargo 2003).
Fluoride Toxicity in Animals by Rakesh Ranjan, Amita Ranjan